{"product_id":"n-acetyl-semax-amidate-20mg-biolongevity-labs","title":"N-Acetyl Semax Amidate X 20mg","description":"\u003cp\u003eDescription--N‑Acetyl Semax Amidate is a stabilized synthetic heptapeptide derived from the ACTH(4‑7) fragment, structurally defined as Ac‑Met‑Glu‑His‑Phe‑Pro‑Gly‑Pro‑NH₂. It incorporates N‑terminal acetylation and C‑terminal amidation, modifications that enhance resistance to enzymatic degradation and extend biological activity. These structural adaptations increase peptide stability, prolong half‑life (approximately 30 minutes longer than standard Semax), and improve neuroprotective efficacy.\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003eMechanistic Claims\u003c\/strong\u003e\u003c\/p\u003e\n\u003cul\u003e\n\u003cli\u003e\n\u003cstrong\u003eMelanocortin Receptor Modulation:\u003c\/strong\u003e Interacts with MC₄ and MC₅ receptors, influencing pathways linked to cognition, stress response, and neuroprotection.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eNeurotransmitter Regulation:\u003c\/strong\u003e Modulates serotonin and dopamine activity, supporting mood and cognitive processes.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eBDNF Upregulation:\u003c\/strong\u003e Enhances brain‑derived neurotrophic factor (BDNF) expression, promoting neuroplasticity and regeneration.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eStructural Stability:\u003c\/strong\u003e N‑acetylation and amidation confer resistance to enzymatic breakdown, extending biological activity.\u003c\/li\u003e\n\u003cli\u003e\n\u003cstrong\u003eExtended Half‑Life:\u003c\/strong\u003e Demonstrates longer duration of action compared to standard Semax formulations.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003eResearch Applications\u003c\/strong\u003e\u003c\/p\u003e\n\u003cul\u003e\n\u003cli\u003eCognitive enhancement and memory studies\u003c\/li\u003e\n\u003cli\u003eNeuroprotection and neuroregeneration models\u003c\/li\u003e\n\u003cli\u003eStroke recovery and ischemic injury investigations\u003c\/li\u003e\n\u003cli\u003eNeurodegenerative disease research (e.g., Alzheimer’s, Parkinson’s)\u003c\/li\u003e\n\u003cli\u003eStress response and mood regulation studies\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003eN‑Acetyl Semax Amidate Research Insights\u003c\/strong\u003e\u003c\/p\u003e\n\u003cul\u003e\n\u003cli\u003eSynthetic heptapeptide engineered for enhanced stability and efficacy.\u003c\/li\u003e\n\u003cli\u003eStructural modifications (acetylation and amidation) improve resistance to enzymatic degradation.\u003c\/li\u003e\n\u003cli\u003eDemonstrates prolonged half‑life and increased neuroprotective potential compared to standard Semax.\u003c\/li\u003e\n\u003cli\u003ePreclinical studies highlight roles in cognitive enhancement, neurotransmitter modulation, and BDNF upregulation.\u003c\/li\u003e\n\u003cli\u003eInvestigated for therapeutic potential in stroke recovery, neurodegenerative disorders, and stress‑related conditions.\u003c\/li\u003e\n\u003c\/ul\u003e\n\u003cp\u003e\u003c!--EndFragment --\u003e\u003cbr\u003e\u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003eN-Acetyl Semax Research\u003c\/strong\u003e\u003c\/p\u003e\n\u003cp\u003eN-Acetyl Semax is a synthetic heptapeptide demonstrating neuroprotective and cognitive-enhancing properties. Research indicates its potential in preventing amyloid beta aggregation, providing neuroprotection in ischemic conditions, and enhancing learning and memory functions. The peptide achieves these effects through modulation of neurotransmitter systems and regulation of inflammatory responses, while also showing promise in stress response and immune system function.\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003e\u003cem\u003eNeuroprotective and Cognitive Effects\u003c\/em\u003e\u003c\/strong\u003e\u003c\/p\u003e\n\u003cp\u003eSemax has been shown to prevent the formation of amyloid beta (Aβ) complexes with copper ions, which are implicated in Alzheimer’s disease, inhibiting fibrillogenesis and providing protective effects against\u003cspan\u003e \u003c\/span\u003e\u003cspan class=\"whitespace-nowrap\"\u003eneurodegeneration.\u003csup\u003e1\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eThe peptide has additionally been found to exert neuroprotective effects in the context of ischemic brain conditions. It enhances angioprotective, antihypoxic, and neurotrophic activities, which are crucial during the acute period of ischemic stroke. Furthermore, Semax administration has been associated with the modulation of inflammatory responses, promoting anti-inflammatory agents over pro-inflammatory factors, which is beneficial in post-ischemic conditions.\u003csup\u003e2\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eSemax is recognized for its nootropic effects, which include improvements in cognitive functions such as learning and memory. In animal models, Semax has been shown to enhance cognitive recovery in cases of chronic brain ischemia, particularly when used in combination with hopantenic acid.\u003csup\u003e3\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eThe mechanisms underlying the potential cognitive and neuroprotective effects of Semax involve its interaction with various neurotransmitter systems and neurotrophic factors. Semax modulates monoaminergic systems, particularly serotonin and dopamine, which are crucial for mood regulation and cognitive functions. Semax increases serotonin turnover and enhances dopamine release, which can improve learning and memory.\u003csup\u003e4\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eMoreover, Semax has been observed to increase brain-derived neurotrophic factor (BDNF) levels in the basal forebrain, a region associated with cognitive functions, further supporting its role in enhancing cognitive abilities.\u003csup\u003e5\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003e\u003cem\u003eSemax and Alzheimer’s Disease (AD)\u003c\/em\u003e\u003c\/strong\u003e\u003c\/p\u003e\n\u003cp\u003eSemax is an ACTH-like peptide that has shown the ability to form stable complexes with Cu2+ ions. This property is crucial as it helps prevent the formation of Aβ:Cu2+ complexes, which are implicated in the pathogenesis of AD. Studies have demonstrated that Semax can inhibit fibrillogenesis, particularly the formation of oligomeric species, thereby exhibiting anti-aggregating properties.\u003csup\u003e1\u003c\/sup\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003eResearch has indicated that Semax can enhance the survival of cholinergic neurons in the basal forebrain, which are known to degenerate in Alzheimer’s dementia. In vitro studies have shown that Semax can increase the survival rate of these neurons by approximately 1.5-1.7 fold and stimulate the activity of choline acetyltransferase, an enzyme critical for acetylcholine \u003cspan class=\"whitespace-nowrap\"\u003esynthesis.\u003csup\u003e6\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eWhile the exact implications of Semax in Alzheimer’s disease require further clarification, its ability to modulate Aβ aggregation and support neuronal survival presents a promising avenue for therapeutic development. Continued research is necessary to explore the full potential of Semax, including its delivery methods and efficacy in clinical settings.\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cem\u003e\u003cstrong\u003e\u003cspan class=\"whitespace-nowrap\"\u003eSemax and Stress Response\u003c\/span\u003e\u003c\/strong\u003e\u003c\/em\u003e\u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eSemax has demonstrated significant immunomodulatory properties in models of “social” stress. It effectively restores cellular and humoral immune responses, as well as the phagocytic activity of neutrophils, indicating its potential as an immune corrector under stress conditions\u003csup\u003e7\u003c\/sup\u003e. Additionally, Semax helps in normalizing the levels of pro- and anti-inflammatory cytokines, such as IL-1β, IL-6, and TNF-α, which are typically elevated under stress.\u003csup\u003e8\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eResearch indicates that Semax can block the opioid form of stress-induced analgesia (SIA) without affecting behavioral changes in rats exposed to acute stressors such as inescapable foot shock and forced cold-water swim stress. This suggests that while Semax can modulate pain sensitivity under stress, it does not alter stress-induced behavioral responses.\u003csup\u003e9\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cspan class=\"whitespace-nowrap\"\u003eIn conditions of informational and social stress, Semax exhibits stress-protective effects by reducing stress-induced physiological changes, such as adrenal hypertrophy and gastric mucosa lesions. It also shows antioxidant properties by decreasing lipid peroxidation in immunocompetent organs like the thymus and spleen, thereby mitigating stress-induced immune dysfunction.\u003csup\u003e10\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cem\u003e\u003cstrong\u003eImmune and Vascular System Modulation\u003c\/strong\u003e\u003c\/em\u003e\u003c\/p\u003e\n\u003cp\u003eIn a study on rat brain focal ischemia, Semax was found to enhance the expression of genes related to the immune system, particularly those encoding immunoglobulins and chemokines, which are crucial for immune cell activity and \u003cspan class=\"whitespace-nowrap\"\u003emobility.\u003csup\u003e11\u003c\/sup\u003e\u003cspan\u003e \u003c\/span\u003eThis immunomodulatory effect was further supported by research demonstrating Semax’s ability to restore cellular and humoral immune responses and phagocytic activity of neutrophils under “social” stress conditions, indicating its potential as an effective immune corrector.\u003csup\u003e7\u003c\/sup\u003e\u003c\/span\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003eIn addition to its effects on the immune system, Semax also modulates the vascular system. In ischemic conditions, Semax altered the expression of genes associated with the development and migration of endothelial tissue, smooth muscle cell migration, hematopoiesis, and vasculogenesis.\u003csup\u003e11\u003c\/sup\u003e\u003c\/p\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003eReferences\u003c\/strong\u003e\u003c\/p\u003e\n\u003col\u003e\n\u003cli\u003eSciacca, M., Naletova, I., Giuffrida, M., \u0026amp; Attanasio, F. (2022). Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. \u003cem\u003eACS Chemical Neuroscience\u003c\/em\u003e, 13, 486 – 496. \u003ca href=\"https:\/\/doi.org\/10.1021\/acschemneuro.1c00707\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.1021\/acschemneuro.1c00707\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eMiasoedova, N., Skvortsova, V., Nasonov, E., Zhuravleva, E., Grivennikov, I., Arsen’eva, E., \u0026amp; Sukhanov, I. (1999). [Investigation of mechanisms of neuro-protective effect of semax in acute period of ischemic stroke].. \u003cem\u003eZhurnal nevrologii i psikhiatrii imeni S.S. Korsakova\u003c\/em\u003e, 99 5, 15-9.\u003cspan\u003e \u003c\/span\u003e\u003ca href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/17603664\/\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/pubmed.ncbi.nlm.nih.gov\/17603664\/\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eKirchev, V. (2023). Cognitive function restoration in rats with chronic brain ischemia using Semax and hopantenic acid comprehensive administration. \u003cem\u003eJournal of Education, Health and Sport\u003c\/em\u003e. \u003ca href=\"https:\/\/doi.org\/10.12775\/jehs.2023.13.04.046\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.12775\/jehs.2023.13.04.046\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eEremin, K., Kudrin, V., Grivennikov, I., Miasoedov, N., \u0026amp; Rayevsky, K. (2004). Effects of Semax on Dopaminergic and Serotoninergic Systems of the Brain. \u003cem\u003eDoklady Biological Sciences\u003c\/em\u003e, 394, 1-3. \u003ca href=\"https:\/\/doi.org\/10.1023\/B:DOBS.0000017114.24474.40\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.1023\/B:DOBS.0000017114.24474.40\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eDolotov, O., Karpenko, E., Seredenina, T., Inozemtseva, L., Levitskaya, N., Zolotarev, Y., Kamensky, A., Grivennikov, I., Engele, J., \u0026amp; Myasoedov, N. (2006). Semax, an analogue of adrenocorticotropin (4–10), binds specifically and increases levels of brain‐derived neurotrophic factor protein in rat basal forebrain. \u003cem\u003eJournal of Neurochemistry\u003c\/em\u003e, 97. \u003ca href=\"https:\/\/doi.org\/10.1111\/j.1471-4159.2006.03658.x\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.1111\/j.1471-4159.2006.03658.x\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eGrivennikov, I., Dolotov, O., Zolotarev, Y., Andreeva, L., Myasoedov, N., Leacher, L., Black, I., \u0026amp; Dreyfus, C. (2008). Effects of behaviorally active ACTH (4-10) analogue – Semax on rat basal forebrain cholinergic neurons.. \u003cem\u003eRestorative neurology and neuroscience\u003c\/em\u003e, 26 1, 35-43.\u003cspan\u003e \u003c\/span\u003e\u003ca href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/18431004\/\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/pubmed.ncbi.nlm.nih.gov\/18431004\/\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eYasenyavskaya, A., Samotrueva, M., Myasoedov, N., \u0026amp; Andreeva, L. (2022). The experimental study of the immunomodulating action of Semax and Selank on the model of „social” stress. \u003cem\u003eEuropean Pharmaceutical Journal\u003c\/em\u003e, 69, 54 – 60. \u003ca href=\"https:\/\/doi.org\/10.2478\/afpuc-2022-0004\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.2478\/afpuc-2022-0004\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eYasenyavskaya, A., Samotrueva, M., Tsibizova, A., Bashkina, O., Andreeva, L., \u0026amp; Myasoedov, N. (2022). Influence of Semax on the Level of Pro- and Anti-Inflammatory Cytokines in Conditions of “Social” Stress. \u003cem\u003eCurrent Drug Therapy\u003c\/em\u003e. \u003ca href=\"https:\/\/doi.org\/10.2174\/1574885517666220831155411\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.2174\/1574885517666220831155411\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eGlazova, N., Myasoedov, N., Limborska, S., Dergunova, L., Kamensky, A., Andreeva, L., Sebentsova, E., Vilensky, D., Manchenko, D., \u0026amp; Levitskaya, N. (2023). Effects of Semax in the Models of Acute Stress. \u003cem\u003eРоссийский физиологический журнал им  И  М  Сеченова\u003c\/em\u003e. \u003ca href=\"https:\/\/doi.org\/10.31857\/s0869813923010053\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.31857\/s0869813923010053\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eSamotrueva, M., Yasenyavskaya, A., Murtalieva, V., Myasoedov, N., \u0026amp; Andreeva, L. (2019). INFLUENCE OF SEMAX ON THE INTENSITY OF LIPID PEROXIDATION IN IMMUNOCOMPETENT ORGANS IN THE CONDITIONS OF “SOCIAL” STRESS. , 19, 188-191. \u003ca href=\"https:\/\/doi.org\/10.17816\/maj191s1188-191\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.17816\/maj191s1188-191\u003c\/a\u003e.\u003c\/li\u003e\n\u003cli\u003eMedvedeva, E., Dmitrieva, V., Povarova, O., Limborska, S., Skvortsova, V., Myasoedov, N., \u0026amp; Dergunova, L. (2014). The peptide semax affects the expression of genes related to the immune and vascular systems in rat brain focal ischemia: genome-wide transcriptional analysis. \u003cem\u003eBMC Genomics\u003c\/em\u003e, 15, 228 – 228. \u003ca href=\"https:\/\/doi.org\/10.1186\/1471-2164-15-228\" rel=\"noopener\" target=\"_blank\"\u003ehttps:\/\/doi.org\/10.1186\/1471-2164-15-228\u003c\/a\u003e.\u003c\/li\u003e\n\u003c\/ol\u003e\n\u003cp\u003e\u003cbr\u003eLabel--\u003cstrong\u003ePeptide Information\u003c\/strong\u003e\u003c\/p\u003e\n\u003ctable class=\"bg-bg-100 min-w-full border-separate border-spacing-0 text-sm leading-[1.88888] whitespace-normal\" style=\"width: 99.9363%;\"\u003e\n\u003cthead class=\"border-b-border-100\/50 border-b-[0.5px] text-left\"\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003cth class=\"text-text-000 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003eProperty\u003c\/th\u003e\n\u003cth class=\"text-text-000 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003eValue\u003c\/th\u003e\n\u003c\/tr\u003e\n\u003c\/thead\u003e\n\u003ctbody\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003ePeptide Sequence\u003c\/td\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003eAc-Met-Glu-His-Phe-Pro-Gly-Pro-NH2\u003c\/td\u003e\n\u003c\/tr\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003eMolecular Formula\u003c\/td\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003eC37H51N9O10S\u003c\/td\u003e\n\u003c\/tr\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003eMolecular Weight\u003c\/td\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003e813.9 g\/mol\u003c\/td\u003e\n\u003c\/tr\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003eCAS Number\u003c\/td\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003e80714-61-0\u003c\/td\u003e\n\u003c\/tr\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003ePubChem CID\u003c\/td\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003e9811102\u003c\/td\u003e\n\u003c\/tr\u003e\n\u003ctr class=\"[tbody\u0026gt;\u0026amp;]:odd:bg-bg-500\/10\"\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 26.2032%;\"\u003eSynonyms\u003c\/td\u003e\n\u003ctd class=\"border-t-border-100\/50 [\u0026amp;:not(:first-child)]:-x-[hsla(var(--border-100) \/ 0.5)] border-t-[0.5px] px-2 [\u0026amp;:not(:first-child)]:border-l-[0.5px]\" style=\"width: 73.2837%;\"\u003eSemax, 80714-61-0, ACTH (4-7), Pro-Gly-Pro-, MEHFPGP, Met-Glu-His-Phe-Pro-Gly-Pro\u003c\/td\u003e\n\u003c\/tr\u003e\n\u003c\/tbody\u003e\n\u003c\/table\u003e\n\u003cp\u003e \u003c\/p\u003e\n\u003cp\u003e\u003cstrong\u003eSemax Peptide Structure\u003c\/strong\u003e\u003c\/p\u003e\n\u003cp\u003e\u003cimg data-opt-id=\"1529272240\" decoding=\"async\" src=\"https:\/\/pubchem.ncbi.nlm.nih.gov\/image\/imgsrv.fcgi?cid=9811102\u0026amp;t=l\" alt=\"Semax.png\" title=\"N-Acetyl Semax Amidate (20mg) 3\"\u003e\u003c\/p\u003e\n\u003cp\u003eSource:\u003cspan\u003e \u003c\/span\u003e\u003ca href=\"https:\/\/pubchem.ncbi.nlm.nih.gov\/compound\/9811102#section=2D-Structure\" rel=\"noopener\" target=\"_blank\"\u003ePubChem\u003c\/a\u003e\u003c\/p\u003e\n\u003cstyle type=\"text\/css\"\u003e\n        td {border: 1px solid #cccccc;}\n        br {mso-data-placement: same-cell;}\n        .bold {font-weight: bold;}\n    \u003c\/style\u003e\n\u003cp\u003e\u003cbr\u003eDosage--\u003cstrong\u003eThis PRODUCT IS INTENDED AS A RESEARCH CHEMICAL ONLY.\u003c\/strong\u003e\u003cspan\u003e This designation allows the use of research chemicals strictly for in vitro testing and laboratory experimentation only. All product information available on this website is for educational purposes only.  This product should only be handled by licensed, qualified professionals. This product is not a drug, food, or cosmetic and may not be misbranded, misused or mislabeled as a drug.\u003c\/span\u003e\u003c\/p\u003e","brand":"Biolongevity Labs","offers":[{"title":"Default Title","offer_id":47947417223323,"sku":"NASEM-20MG","price":8222.0,"currency_code":"INR","in_stock":true}],"thumbnail_url":"\/\/cdn.shopify.com\/s\/files\/1\/0613\/3694\/4795\/files\/NASEM-20MG-n-acetyl-semax-amidate-20mg-RET.jpg?v=1770933379","url":"https:\/\/fmihealth.com\/products\/n-acetyl-semax-amidate-20mg-biolongevity-labs","provider":"FMI health","version":"1.0","type":"link"}